Options to question: 1. plasma/filtrate 2. reduction/increase 3. increase/decrease 4. myogenic/cardiogenic 5. constrict/
Posted: Sun Jul 10, 2022 4:25 pm
Options to question:
1. plasma/filtrate
2. reduction/increase
3. increase/decrease
4. myogenic/cardiogenic
5. constrict/dilate
6. narrow/enlarge
7. reduce/increase
8. glomerulotubular/tubuloglomerular
9. decrease/increase
10. decrease/increase
11. more/less
12. increase/decrease
(a) Define GFR; how would you expect dehydration to affect this rate? (b) Define autoregulation and its two negative feedback mechanisms. How would they respond to someone experiencing high blood pressure, like Jonjei? (I strongly recommend you watch this other video before answering part B of this question!) A. GFR is defined as the amount of [Select] formed by both kidneys in 1 minute. Dehydration leads to a(n) [Select] in the amount of water in the blood plasma. Less blood plasma would be filtered through the kidneys, and so the GFR would [Select] B. Autoregulation is the ability of the kidneys to maintain GFR over a wide range of systemic blood pressure changes using the two negative feedback mechanisms below. Both of these mechanisms would respond to preserve Jonjei's GFR. o The [Select]
o The [Select] mechanism is related to the amount of stretch a particular blood pressure exerts on the walls of the kidney's blood vessels. Jonjei's blood pressure is elevated, which would increase the blood pressure exerted on the kidney's blood vessels. This in turn would cause those vessels to [Select] or [Select] , to [Select] blood flow through the glomerulus. This mechanism would lower GFR. [Select] feedback is the second autoregulation mechanism for maintaining GFR; it involves the cells of the macula densa monitoring the sodium chloride concentration in the blood. Jonjei's high blood pressure would initially increase the pressure in the glomerulus, and cause the GFR to [Select] ✰. The macula
[Select] û The macula densa would detect more sodium and chloride ions, and would respond by signaling the juxtaglomerular cells to [Select] renin release. This would result in [ Select] angiotensin- II and the contraction of the efferent arteriole, which would [Select] the GFR. In Jonjei's case, this mechanism would be effective.
1. plasma/filtrate
2. reduction/increase
3. increase/decrease
4. myogenic/cardiogenic
5. constrict/dilate
6. narrow/enlarge
7. reduce/increase
8. glomerulotubular/tubuloglomerular
9. decrease/increase
10. decrease/increase
11. more/less
12. increase/decrease
- Options To Question 1 Plasma Filtrate 2 Reduction Increase 3 Increase Decrease 4 Myogenic Cardiogenic 5 Constrict 1 (52.36 KiB) Viewed 36 times
- Options To Question 1 Plasma Filtrate 2 Reduction Increase 3 Increase Decrease 4 Myogenic Cardiogenic 5 Constrict 2 (44.5 KiB) Viewed 36 times
- Options To Question 1 Plasma Filtrate 2 Reduction Increase 3 Increase Decrease 4 Myogenic Cardiogenic 5 Constrict 3 (46.76 KiB) Viewed 36 times
o The [Select] mechanism is related to the amount of stretch a particular blood pressure exerts on the walls of the kidney's blood vessels. Jonjei's blood pressure is elevated, which would increase the blood pressure exerted on the kidney's blood vessels. This in turn would cause those vessels to [Select] or [Select] , to [Select] blood flow through the glomerulus. This mechanism would lower GFR. [Select] feedback is the second autoregulation mechanism for maintaining GFR; it involves the cells of the macula densa monitoring the sodium chloride concentration in the blood. Jonjei's high blood pressure would initially increase the pressure in the glomerulus, and cause the GFR to [Select] ✰. The macula
[Select] û The macula densa would detect more sodium and chloride ions, and would respond by signaling the juxtaglomerular cells to [Select] renin release. This would result in [ Select] angiotensin- II and the contraction of the efferent arteriole, which would [Select] the GFR. In Jonjei's case, this mechanism would be effective.