A neurotransmitter increases chloride permeability through an ligand-gated (ionotropic) receptor channel but other ion p

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A Neurotransmitter Increases Chloride Permeability Through An Ligand Gated Ionotropic Receptor Channel But Other Ion P 1 (16.51 KiB) Viewed 69 times

A Neurotransmitter Increases Chloride Permeability Through An Ligand Gated Ionotropic Receptor Channel But Other Ion P 2 (37.03 KiB) Viewed 69 times

A Neurotransmitter Increases Chloride Permeability Through An Ligand Gated Ionotropic Receptor Channel But Other Ion P 3 (23.5 KiB) Viewed 69 times

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A Neurotransmitter Increases Chloride Permeability Through An Ligand Gated Ionotropic Receptor Channel But Other Ion P 5 (12.83 KiB) Viewed 69 times

A neurotransmitter increases chloride permeability through an ligand-gated (ionotropic) receptor channel but other ion permeabilities are not affected. If the dendrites of the post-synaptic cell have a resting Em of -70mV and a chloride Nernst Equilibrium potential (ECI-) of -90mV, the neurotransmitter will cause a net diffusion of chloride the postsynaptic neuron's dendrites, which will cause a(n) A out of, EPSP a into; EPSP Ocinto; IPSP Dout of: IPSP
Gabby has an incredibly rare condition in which she can sense and perceive most sensations, but can not feel pain. Based on what you know about the nervous system, which of the following would be a viable hypothesis for why Gabby doesn't feel pain. OA. Her pacinian corpuscles are hyper-activated and overwhelm the sensory input to her somatosensory cortex OB. Her nocioceptors are in a state of constant tonic adaptation, so they can not communicate changes in pain sensation OC. Her primary somatosensory cortex never developed, so she can not process the pain sensations OD. There is a mutation in voltage-gated Na+ channels that are specific to nocioceptors, which prevents the nocioceptors from firing action potentials.
Which of the following is true of the Nodes of Ranvier? OA. They are located along the axon of a neuron and have a high concentration of voltage-gated Ca++ channels. OB. They are located on neuronal axons, at the gaps between myelination. There is a high concentration of voltage gated Na+ channels and voltage gated K+ channels which allow for the production of action potentials at the Nodes of Ranvier. C. They are produced by microglia in the central nervous system and satellite cells in the peripheral nervous system. D. They are areas of myelination which prevent loss of charge across the plasma membrane of the neuron. E. They increase the loss of charge along the plasma membrane of a neuron.
The axon terminal membrane has a high density of neurotransmitter is linked to the of A. voltage-gated sodium; efflux; sodium B. voltage-gated potassium; efflux; potassium C. voltage-gated potassium; influx; potassium sodium; influx; sodium OD.voltage-gated OE. voltage-gated calcium; influx; calcium F. voltage-gated calcium; efflux; calcium ions. channels, and the release of
Intensity of a sensory stimulus is encoded in the following way(s): A increasing the amplitude of deflection of the action potential B. differences in firing rate frequencies C. activating different types of receptors D. changing the number of receptors activated