- Potassium 7 5 Meq L Arterial Blood Ph Is 7 20 Paco 20 Mmhg Nahco3 10 Meq L Pao2 66 Mmhg On Room Air Hemodialysis H 1 (143.5 KiB) Viewed 63 times
- Potassium 7 5 Meq L Arterial Blood Ph Is 7 20 Paco 20 Mmhg Nahco3 10 Meq L Pao2 66 Mmhg On Room Air Hemodialysis H 2 (143.5 KiB) Viewed 63 times
- Potassium 7 5 Meq L Arterial Blood Ph Is 7 20 Paco 20 Mmhg Nahco3 10 Meq L Pao2 66 Mmhg On Room Air Hemodialysis H 3 (212.41 KiB) Viewed 63 times
potassium 7.5 mEq/L. Arterial blood pH is 7.20, Paco, 20 mmHg, NaHCO3 10 mEq/L, PaO2 66 mmHg on room air. Hemodialysis has become necessary. 17 Hood purperan du 13 mol functioning vidneys 3. What current signs and symptoms (those found in Paragraph B) would you attribute to Mrs. S.'s elevated potassium? (Choose the correct answer) a. Electrocardiogram changes, weakness, dyspnea, and nausea b. Pruritis, S3 gallop, pitting edema, and weight gain C. Thirst, hypertension, peaked T waves, oliguria, and weight gain d. Pruritis, acidosis, S3 gallop, weight gain, and nausea. Ten days later, Mrs. S. is feeling better. She is able to eat. Mrs. S.’s vital signs are closer to her normal baseline. Her edema, both peripheral and pulmonary, is gone, as are weakness and pruritis. Her blood urea nitrogen is 40 mg/dl and creatinine 6.0 mg/dl. Mrs. S.'s metabolic acidosis is controlled. Her urine output has increased dramatically and ranges between 100 and 150 ml/hr. Seven months post discharge from the hospital, her blood urea nitrogen plateaued at 27 mg/dl and her creatinine at 1.4 mg/dl. Mrs. S. has finally recovered. 4. Using the lab results data and by comparing how the lab results changed over time, list a few of Mrs. S.'s acid-base imbalances and how her body compensated. a
Normal MAP: 70-100mmHg, Pt: 50mming Normal HR (tying): 60-100b/min Pt: 95 blm •Norma) BP (est): Poda 120/80 a Normal ir (standing).goes uplo-5 bpm PH: 100 blm Murnal BP (stand): 120/80 Pt (stand): 134178 Pt: 138/84 e Case Study #2 Pressur gettings aren't enough salah sebelum Mean Arterial Ie are stable shook Vincava Mrs. S. is a 69-year-old critically ill woman who experienced a major hypotensive episode, secondary to gastrointestinal hemorrhage and hypovolemic shock. y low blood Ly hervy bleeding in Lyou plasma means Mrs. S. had a massive gastrointestinal bleed last night. The bleeding has stopped, blad / os her causng Pressure) fell to less than 50' for more than 40 minutes. Mrs. S. has no unexpected complaints. She is very relieved that her stomach ulcer has stopped bleeding. Otherwise, she has only minor discomforts associated with treatment. Mrs. S.'s lying heart rate is 95 and blood pressure 138/84; standing heart rate is 100 and blood fleeting vertigo Seinschone is without jugular venous distever qune 10 and experiences only rate is 20 and unlabored, and her lungs have transitory sales that clear with coughing. Hun taj SOKASTIS swelling lower body Unfortunately, her urine output has been sluggish (15-20 ml/hr) overnight and has fallen to 10 ml/hr for the last 4 hours. The urine is a clear yellow. Urine specific gravity is 1.010, sodium 50 mEq/L, serum blood urea nitrogen 43 mg/dl, creatinine 1.3 mg/dl. weinidogens 1. The probable reason for a drop in GFR and subsequent oliguria while Mrs. S. was bleeding was: (Choose the correct answer) Lylow urine output a. Rapid transfusions b. Systemic acidosis c. Systemic hypoxia d. Lack of renal perfusion 2. Mrs. S.'s renin mechanism and the release of two other hormones (aldosterone and Anti-diuretic hormone (ADH)) were called into play during her hypovolemic shock episode. Explain how Mrs. S.'s renin mechanism and the action of Aldosterone and ADH protected vital organs such as the brain and heart from experiencing a critical decrease in blood flow. Two days later, Mrs. Sy is a little breathless, irritable, and tired. She complains of thirst, blood pressure 168/92. She has gained 6 kg and has sher heart rate is 86, and her, ofter pressing nausea mild pruritis, weakness and shortness of breath. edema around her ankles. seconds Pulses are full and she has an Sheart soundclopa, depend highly labored. She is amounds (isks) Her lungs have fine Pheart failure third of the way up posteriorly. Her respiration is weak and lethargic. Mrs. S.'s electrocardiogram shows tall, peaked I waves and yocardial inf pretiun widening intervals. Her serum blood urea nitrogen is 180 mg/d1, creatinine 10 mg/di, 28 sign of hyperkalemia B