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the intestines, triggering the loss of electrolytes and water. The of Shigella begins when the pathogen attaches to pathogenesis [Select] cells in the large intestine, stimulating endocytosis of the bacterium, followed by multiplication in host cell. Shigella polymerizes the host's [Select] , propelling itself out of the host cell and into adjacent cells, thus evading the host's immune system. As the bacterium kills host cells, [Select] form in the mucosa and any bacteria that enter the blood from a ruptured abscess are quickly phagocytized and destroyed, making [ Select] a rare complication of shigellosis. is one of a group of colon-dwelling bacteria called coliforms. A few antigens, such as 0157, 0111, H8, and H7, are associated with more severe [ Select] . These strains-known as enterohemorrhagic E. coli -have genes located on transmissible [ Select] for fimbriae, adhesins, and a variety of toxins that enable EHEC to cause severe foodborne disease. The [Select]